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REPORT BY PROFESSOR STEVE H. RUDD
PART II
8. Despite repeated attempts to find chemical, structural or scanned differences, schizophrenics have generally normal brains, except when taking psychiatric drugs:
(a) "In 1978, Philip Seeman at the University of Toronto announced in Nature that this was indeed the case. At autopsy, the brains of twenty schizophrenics had 70 percent more D2 receptors than normal. At first glance, it seemed that the cause of schizophrenia had been found, but Seeman cautioned that all of
the patients had been on neuroleptics prior to their deaths. "Although these results are apparently compatible with the dopamine hypothesis of
schizophrenia in general," he wrote, the increase in D2 receptors might "have resulted from the long-term administration of neuroleptics." [T. Lee, "Binding of 31-1-neuroleptics and 3H-apomorphine in schizophrenic brains," Nature 374 (1978): 897-900.] … A variety of studies quickly proved that the drugs were indeed the culprit. When rats were fed neuroleptics, their D2 receptors
quickly increased in number. [D. Burt, "Antischizophrenic drugs: chronic treatment elevates dopamine receptor binding in brain," Science 196 (1977): 326-27.] … If rats were given a drug that blocked D, receptors, that receptor subtype increased in density. [M. Porceddu, "[3H]SCH 23390 binding sites increase after chronic blockade of d-1 dopamine receptors," European Journal
of Pharmacology 118 (1985): 367-70.] … "Finally, investigators in France, Sweden, and Finland used positron emission topography to study D2-receptor densities in living patients who had never been exposed to neuroleptics, and all reported "no significant differences" between the schizophrenics and "normal controls."" [J. Martinot, "Striatal D2 dopaminergic receptors assessed with positron emission tomography and bromospiperone in untreated
schizophrenic patients," American Journal of Psychiatry 147 (1990): 44-50; L. Farde, "D2 dopamine receptors in neuroleptic-naive schizophrenic patients," Archives of General Psychiatry 47 (1990): 213-19; J. Hietala, "Striatal D2 dopamine receptor characteristics in neurolepticnaïve schizophrenic patients studied with positron emission tomography," Archives of General Psychiatry 51 (1994): 116-23.] - page 76 of Anatomy of an Epidemic, by Dr. Robert Whitaker (2010)
(b) "The low-serotonin hypothesis of depression and the high-dopamine hypothesis of schizophrenia had always been the twin pillars of the chemical- imbalance theory of mental disorders, and by the late 1980s, both had been found wanting. Other mental disorders have also been touted to the public as diseases caused by chemical imbalances, but there was never any evidence to support those claims." - Ibid, page 77
(c) "There is no compelling evidence that a lesion in the dopamine system is a primary cause of schizophrenia" - page 392 of Molecular Neuropharmacology, by E.Nestler & S.Hyman (2002)
9. No amount of repetition is sufficient - Schizophrenia, with or without paranoia, is a voluntary behaviour choice, motivated by real or imagined present or expected personal benefit, and assuredly not a disease - and therefore cannot be treated. It can be punished with sufficient severity (financial and deprivation of liberty) which will make it an unattractive lifestyle choice for the perpetrator.
10. By blocking this happening, from the viewpoint of society, Panigrahi, Odesanya and Kingham are co-conspirators. These shrinks are accessories
before, during and after the fact (this, per Bhardwaj) and I believe deserve harsh condemnation.
For the greater good, any jurist would agree that Panigrahi (identified in Appendix C below) himself must be locked up immediately. His "paranoid schizophrenia" will then miraculously disappear. Remove this financial motive, and the antisocial, deviant, selfish and damaging behaviour will cease.
Such a shame corporal punishment is currently out of vogue, or accelerated
cure could be effected.
Prof. S. Rudd
(Beware of Quacks)
Appendix A ---- DSM-V Classification (for use by Quacks)
F20-F29 Schizophrenia, schizotypal and delusional,
and other non-mood psychotic disorders
F20 Schizophrenia
F20.0 Schizophrenia with paranoia
F20.1 Hebephrenic schizophrenia
F20.2 Catatonic schizophrenia
F20.3 Undifferentiated schizophrenia
F20.4 Postschizophrenic depression
F20.5 Residual schizophrenia
F20.6 Simple schizophrenia
F20.8 Other schizophrenia
F20.9 Schizophrenia, unspecified
F21 Schizotypal disorder
F22 Persistent delusional disorders
F22.0 Delusional disorder
F22.8 Other persistent delusional disorders
F22.9 Persistent delusional disorder, unspecified
F23 Acute and transient psychotic disorders
F23.0 Acute polymorphic psychotic disorder without symptoms of schizophrenia F23.1 Acute polymorphic psychotic disorder with symptoms of schizophrenia F23.2 Acute schizophrenia-like psychotic disorder
F23.3 Other acute predominantly delusional psychotic disorders
F23.8 Other acute and transient psychotic disorders
F23.9 Acute and transient psychotic disorder, unspecified
F24 Induced delusional disorder
F25 Schizoaffective disorders
F25.0 Schizoaffective disorder, manic type
F25.1 Schizoaffective disorder, depressive type
F25.2 Schizoaffective disorder, mixed type
F25.8 Other schizoaffective disorders
F25.9 Schizoaffective disorder, unspecified
F28 Other nonorganic psychotic disorders
F29 Unspecified nonorganic psychosis
Appendix B ---- Reference Material to support Clinical Diagnosis
https://www.theconservativelibertariansociety.com
https://shanpanigrahi.co.uk
http://alturl.com/kx7ma http://alturl.com/nuvq9 https://www.thelibertariandemocrats.com/forum https://www.theallurementofrealityinreview.com http://pub46.bravenet.com/forum/3871902446/ http://vishistaadvaitavedanta.bravesites.com/blog
http://alturl.com/gkwkf http://alturl.com/3un5d https://towardsknowledgeforworldconservation.com https://twitter.com/ShantanuPanigr8 https://www.facebook.com/shantanu.panigrahi.10 https://theconservativelibertarianpartyoftheunitedkingdom.com https://the-conservative-libertarian-party-of-usa.odoo.com https://t.co/Y4A0lmdxGr https://t.co/6dXxB3hTZV
https://t.co/88748B8HEk http://alturl.com/p4nho https://www.bing.com/search?q=%22Dr+Shantanu+Panigrahi%22 https://groups.google.com/d/msg/alt.politics/UOpfj3x7Fec/d2VwpvZ3AQAJ https://narkive.com/VT9xrlho https://narkive.com/ZE507Zte https://narkive.com/MPLIJFCX https://narkive.com/Pu5jAiOd https://shantanup.wordpress.com (at archive.org)
https://archive.org/details/@shantanu858
Appendix C ---- The Perpetrator/Subject
Name. "Doctor" Shantanu (Shan) Panigrahi
NHS No. 6284771487 Hospital No. R570808AD National Insurance No. YZ330724D
UK Passport 522465108 (naturalised 1/4/1984)
UK Phones: (+44)01634 379604, (+44)07967 789619
Addresses: Room D, First Floor Basement, 3 Hoath Lane, Wigmore, Gillingham, Kent, ME8 0SL, UK, also Plot 2457/1 Gourinagar, Bhubaneswar 751002, Odisha, India also Rashmi Niwas, A13/3 Kalindi Housing Estate, N. 24 Parganas, S. Dum Dum, Kolkata 700089, W.Bengal, India
Known emails:
shanpanigrahi@yahoo.co.uk,
shanpanigrahi3000@gmail.com,
shantanupanigrahi@aol.com,
shantanupanigrahi@yahoo.com,
panigrahi@gmail.com,
shanpanigrah5000@outlook.com,
catlovers@hotmail.co.uk,
aateurope2@gmail.com Dates of Birth: January 15 and August 8 1957, but other dates depending on benefit or waiver is being claimed; including as "Shan Praharaj'; Rupa Panigrahi September 19 1990
Barclays Current Account Mr S Panigrahi & Mrs R R Panigrahi 20-54-11 90098086 and Savings H169296PAN TCLP-UK-VOPA (and paypal) 09-01-27 11199612 Natwest 60-17-44 Debit Card 4751 2902 3982 1523 exp 11/24
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