Although research in the 1990s hinted at the ability to induce torpor in animals by injection of blood taken from a hibernating animal, further research has been unable to reproduce this phenomenon. Despite the inability to induce torpor, there are
substances in the blood of hibernators that can lend protection to organs for possible transplant. Researchers were able to prolong the life of an isolated pig's heart with an HIT.[36] This may have potentially important implications for organ transplant,
”

smartwatch magnetically releases hibernation induction trigger https://www.annalsthoracicsurgery.org/article/S0003-4975(97)00631-0/fulltext “HIT” (or maintenance) chemicals if it detects 1 per 1000 likeliness of cardiovascular event that hour,
interestingly it may be possible to combine this benefit with a mild wake-up chmical so the person is able to do something other than pass out in response. bodyside and well as brainside varied chemicalcs could also contribute to heightening surviviving
and normalcy after cardiovascular event; bodyside only “HIT” (Hibernation induction trigger (HIT) obtained from serum of certain winter-hibernating mammals, such as woodchucks, 13-lined ground squirrels, brown cave bats, and black bears, can induce
hibernation in these animals, even when summer active) At humans, with a smartwatch drug depot this could be continuouous low level medication for those projected to be at cardiovascular risk without impairing mental function. HIT studies are from 1997
and could be updated to see if it is possible to make RNA drugs and peptides that do the same things as the HIT chemicals, thought at the time to be opiods (like opiate peptides)

As a technologogy to get rid of heart attacks, HIT chemicals could be made to circulate at the body during the first 4 hours (most lethal), as well as during winter. Perhaps bodyside only would do it, leaving people’s minds fully functional in the AM.
Also, the article mentions opiate receptors as HIT life-preserving drug receptors. I read there are more than 100 neurotransmitters, so perhaps there are other non opioid HIT chemicals that can be found with serum between lab mammals experiments.

Also, they found a primate (nearer human biochemistry, mRNA) the fat-tailed dwarf lemur that hibernates up to 7 months a year, along with opiate HIT, these lemurs may have other serum chemical systems that could benefit humans, particularly at redicing
cardiovascular events or making them less serious.

Along with the article URL, that concentrates on treatmentless ischemic hearts having about about 30% their original capacity on survival, and 15 minute pretreated (bear serum or opiates) hearts having 69-80% original capacity this brings up the
possibility of using HIT hibernation chemicals to measurably reduce the effects of strokes or even routine surgical anesthesia on mental functioning.

Under hibernation, Wikipedia mentions sharks can go three hours of ischemia, so shark serum could also be tested on cardiac and strokelike ischemia to find new drugs that minimize the effects of heart attacks and strokes.

As a possible technological improvement to the 1997 HIT paper, cell penetrating peptides could concentrate all of the traversal of active drugs (Bear serum simililar chemicals, opiate peptides) to specific organs, for example, heightening concentration
at the heart and vasculature, making these drugs more dose efficient and possibly better tolerated.

A one dose approach to a drug that increases the survivability of cardiovascular events (heart attacks) is: tail squiggle gene therapy, photoactive installation just at the heart: Describing that, basically receptors, like opiate receptors have receptive
components (one mental reference is g coupled protein receptor swirl fingers) as well as a base area (before the fingers) some authors call the tail that does not directly glom the the thing of interest. This tail area can however have a custom peptide
or protein made, that gloms to it, that causes the actual receptor part (fingers) to be more or less sensistive. So engineering a peptide that causes heart opiate receptors (or bear hibernation serum chemical receptors) to be unusually easy to activate
and causes them to stay activated could cause continuous “on” status just from normal circulating levels of endogenous (natural) chemicals as they are primed to prefer to be on. One way to get the tails to have the perma-on peptide attached to them,
or, perhaps more optimally, just be developed from stem cells with the “easy on” protein sequence and conformation conformation part of their makeup is is phototherapy gene therapy: Using published methods gene therapy only takes (does its thing)
where tissues are illuminated, and at a person, cofocal lighting lights up the heart for the duration of the gene therapy installation. That causes the opiate receptors (or also bear hibernation heart chemical survival receptors) to be the only things
at the body illuminated/eligible, at that particular treatment, for updating with the new genetic form.

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All technologies, ideas, and inventions of Treon Sebastian Verdery are public domain at JUly 8,2023AD and previously, as well as after that date

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