XPost: alt.animals.ethics.vegetarian, alt.food.vegan, alt.food.vegan.science XPost: alt.food.fast-food, alt.survival

On Tue, 17 Sep 2019 01:32:56 -0000 (UTC), "Leroy N. Soetoro" wrote:

<https://hotair.com/archives/jazz-shaw/2019/09/11/plant-based-burgers-arent-better-eating/>
The title question is being asked by Kate Bernot at The Takeout, and it’s
a good one. Before anyone starts imagining a totally meatless future >(speaking of horror shows), we should figure out why so many people are >flocking to plant-based “meat” like the Impossible Whopper and its
cousins. Since experts are quickly concluding that these products are no >healthier for you (or for the environment) than their traditional animal- >based counterparts, what’s the attraction making these offerings
sustainable?

I suspect the mass appeal is just more "green" AOC whacko followers
believing their own BS.

BUT
(Short version: meat, especially red meat, and sugar are suspect as
promoting the inflammation that cancer and other diseases of age need
to develop.)

(Longer but still extremely abridged version:)
An article in New Scientist claims Porphyromonas gingivalis, the
bacteria that cause gum disease, is implicated in "lifestyle diseases"
that often come on with age including heart disease, Alzheimer's, type
2 diabetes, strokes, rheumatoid arthritis, Parkinson's, and some
cancers. "DNA sequencing has revealed bacteria in places they were
never supposed to be, manipulating inflammation in just the ways
observed in these diseases." The claim is p. gingivalis promotes
inflation, something the body machine normally tries to reduce, and
that inflammation is required by those diseases.

"Most pathogens try to block or avoid inflammation, which normally
kills them before it shuts down again. Starting in our 30s and 40s,
this shutdown begins failing, leading to the chronic inflammation
involved in diseases of ageing. No one knows why.
"P. gingivalis may have a hand in it. It actually perpetuates
inflammation by producing molecules that block some inflammatory
processes, but not all of them, says Caroline Genco of Tufts
University in Massachusetts. The resulting weakened inflammation never
quite destroys the bacteria, but keeps trying, killing your own cells
in the process. The debris is a feast for P. gingivalis, which, unlike
most bacteria, needs to eat protein.
"The destruction also liberates the iron that bacteria need and
which the body therefore normally keeps locked up. "These bacteria
manipulate their interaction with the host immune response to enhance
their own survival,"

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